The Leading Cause Of Death
Heart disease is the leading cause of death in the civilised world, mostly in the form of coronary heart disease (CHD) which affects the blood vessels supplying blood to your heart. CHD causes your arteries to narrow, therefore reducing the amount of oxygen for your heart.
The conventional widely and conveniently spread believe that cholesterol is the major risk factor for this condition, and the ongoing big clamour about its supposed damage to your heart has resulted in the creation of an enormous market for statins; sold as drugs that reduce cholesterol by blocking the enzyme in your liver that is responsible for making it.
Statins are now among the most widely prescribed drugs on the market, and are the number one profit-maker for the pharmaceutical industry, largely due to relentless and highly successful direct-to-consumer advertising campaigns.
And statins prescriptions and consumption does not abate in spite of about a thousand studies, since the year 2010, that have proved their numerous adverse effects to your health, and their inability to reduce your risk for heart disease, because high cholesterol does NOT increase heart disease risk…
Statins, the most prescribed drugs in the US are also the most controversial ones because of their potential side effects that have become a hot issue.
They have detractors and supporters (the majority). For a minority of cardiologists, statins have to be prescribed for people with high cholesterol levels but who have not had a cardiac event yet and only IF they cannot achieve a cholesterol reduction within a few months of drastic lifestyle change.
An interesting view on cholesterol
Doctors are also divided in those (the majority) who support the imperant theory that there is a linear link between cholesterol levels and heart attack or stroke risk, and feel total cholesterol count should be below 200 if you are healthy and ideally to be brought even lower, below 150, especially if you if you are diagnosed with having heart disease, and in those doctors (the minority) who maintain that we are told a bunch of lies about cholesterol, heart disease and stroke.
In fact, many studies, especially among populations outside the United States, show that there is no link between cholesterol levels, saturated fats, and heart disease.
For example, why ethnic Japanese living in the United States have lower cholesterol levels than those in Japan, yet they have higher heart attack rates’. The same thing has been found with Navajos, Irish immigrants, African nomads, and many others.
It is about 60 years old the prediction that to follow a low-fat, high-carbohydrate diet, low in saturated fats and high in polyunsaturated vegetable oils would make heart disease and atherosclerosis completely disappear by the year 2000.
Unfortunately today, after following the recommended “heart-healthy diet” for decades, the situation has not improved at all, even despite a massive use of statin cholesterol-lowering drugs made available since the past two decades. But in magazine and newspaper ads, the drug companies state that their statin drug lowers heart attack risk by 54 percent and of stroke by 48 when. In reality the risk is lowered just by 1 percent. So why not take much less expensive supplements of vitamins C and E which alone will reduce risk by a much greater percentage. Plus other supplements which combined with a proper diet and exercise dramatically lower your risk -no drugs needed.
But what are Stains and what do they do?
According to supporters and manufacturers statins have allowed the reduction of fatal heart attack by 27 percent in the past 10 years alone.
Statins work by blocking an enzyme in the liver called hdroxy-methilglutaryl-coenzime A reductase, in short HMG-CoA, which is responsible for producing cholesterol. According to manufacturers and enthusiastic supporters, Statins reduce LDL cholesterol total count by 25-30 percent, reduce triglycerides (fats in the blood) by 10 to 25 percent, and mildly raise good HDL cholesterol total count. They are also vasodilators (help expand arteries), prevent plaque deposit from rupturing and improve the important function of endothelial cells (cells of the lining of the arteries) which prevents plaque formation.
More conscientious cardiologists recognise that there are real risks associated with statins, primarily liver and muscle damage but they still keep prescribing them.
Statins work in the liver and raise the level of liver function which is measured testing the enzymes AST (Aspartate Transaminase) and ALT (Alanine Transaminase). These tests indicate if there is inflammation and cell destruction in the liver, if liver functions are increased, that indicates that the statin is causing toxicity.
But “not to worry” say statins supporters, because “liver has amazing power of regeneration unless liver function rises to 2.5-3 times its normal value, and there is such a variety of statins (Simvastin, Atorvastatin, Pravastatin, etc.) that it is unlikely not to find a brand that patients cannot tolerate well because many cases of elevated liver functions are statin-specific”. Well, not exactly.
Liver damage associated with statins include:cirrohosis, hepatitis, and hematochromatosis (iron overload in the blood).
Muscle damage is the other major concern with statins. A form of damage is called Myopathy which causes muscle aches and weakness accompanied by fatigue.
Another more serious form of muscle damage is called Rhabdomyolysis which causes the muscle to breakdown accompanied by with a very painful sensation. Here the dead muscle cells flood into the bloodstream overwhelming the kidneys and causing long-term complications. Unfortunately a study found that 1 in 10 people develop muscle damage even before the symptoms develop.
A Danish study found that those taking statin drugs long term were 4 to 14 times more likely to develop nerve degeneration leading to difficulty walking and painful extremities.
Statins can sap a patient’s vitality by binding testosterone and estrogen thus lowering the libido so hormone levels have to be replenished.
It has already been mentioned, in previous posts, that all the statin drugs dramatically lower heart CoQ10 levels which is needed to keep the heart strong.
Since the introduction of these drugs, there has been a dramatic increase (600 percent) in congestive heart failure — unexplained by any other factors. Because many elderly, especially diabetics, are already low in CoQ10. the effect of consuming the body’s supply of Coenzyme Q10 (CoQ10) theywould need to intake daily large quantities of CoQ10 supplements.
Loss of cognitive functions and even amnesia can show up when starting with statins. Of special concern is the effect on brain function. Besides dementia-like memory loss, cases of personality change, irritability and short temper are being reported. One study expressed a special caution to those with Alzheimer’s disease, who may in fact be more susceptible to the damaging effects of statins, including those with a family history of Alzheimer’s disease.
A recent study,published in the journal Critical Care Medicine revealed that taking statin drugs can drastically reduce your risk of surviving a heart attack. It found that the lower a patient’s cholesterol levels, the higher the risk of dying during the 30-day period following a heart attack.
The patients with low LDL cholesterol levels coupled with low triglyceride levels had a 990 percent increased risk of dying. Patients with LDL-cholesterol levels less than 110 mmg/dl, and triglyceride less than 62.5 mmg/dl were at the highest risk of dying .
Lower LDL levels were linked to a 65 percent increase in mortality: lower triglycerides were linked to a 405 percent increased mortality. Patients with both LDL and triglyceride levels below the threshold levels, had 990 percent increased risk for dying.
Icing on the cake is the elevated risk for developing later-onset diabetes.
But supporters say that the complications from the use of statins affect only a very small percentage of heart disease sufferers while they can treat all heart disease related conditions for up to 50 percent of the patients, and especially a 25 percent reduction in fatal heart attacks so what statins pose is a very acceptable risk.
That is BS, in reality the best statin studies show that for men statin can reduce the risk of preventing a cardiac event only by a paltry 1 to 4 percent, that is, 96 to 99 percent of everyone who takes a statin drug gets no benefit. For women, the numbers are even worse.
A List Of Further Statin Damages:
– Sexual dysfunction
– Immune depression
– increased risk for Lou Gehrig’s disease.
– increased risk for diabetes
Statins can increase your risk of diabetes by y increasing insulin resistance which contributes to chronic inflammation in your body, as we know which is a very serious problem because we know also that inflammation is the hallmark of most diseases.
Ironically the primary reason for taking a cholesterol-reducing drug is to prevent heart disease when just taking statins will cause the insulin resistance which leads to heart disease,
Increased insulin resistance also promotes belly fat, high blood pressure, heart attacks, chronic fatigue, thyroid disruption, and diseases like Parkinson’s, Alzheimer’s, and cancer.
Diabetes risk is also increased by the raising of your blood sugar cause by statins which prevent your liver from making cholesterol. When you eat starches and sugar, the excess sugar goes to your liver, which normally stores it away as cholesterol and triglycerides. But because statins work by preventing your liver from making cholesterol, the liver will return the sugar to your bloodstream, thus raising your blood sugar levels.
– Inhibiting the synthesis of vitamin K2.
Vitamin K2’ is important for the human body because it helps move calcium into your bones and teeth, where you need it. It also removes calcium from your arteries and soft tissues where it shouldn’t be and where it contributes to atherosclerosis. so by reducing K2 presence in the body, statin actually stimulate atherosclerosis and heart failure
The Great Cholesterol Cover-Up
Cholesterol theory is cruel hoax and the use of statins to cure it just a get-rich scheme of Big Pharma, and currently they have a huge market of more than 28 percent of U.S. adults who take a statin drug that is supposed to treat high cholesterol levels.
For the vast majority of people who take them, statins do not prevent nor do they treat heart disease, in fact they cause heart disease. However, the powers-that-be will go to any lengths to convince everyone that statins are effective for treating and preventing heart disease.
The Origin Of The Myth
Rudolph Virchow (1821-1902), a German pathologist found thickening in the arteries in people he autopsied, and attributed it to a collection of cholesterol.
Ancel Keys (1904-2004), a well-known physiologist, published a paper known as the “Seven Countries Study,” which become the basis for all the future scientific support for the Cholesterol Theory. where he linked the consumption of saturated fat to coronary heart disease.
But he conveniently excluded from his study those countries that did not fit with his hypothesis, because they showed a low percentage fat in their diet with a high incidence of death from CHD and those with a high-fat diet and low incidence of CHD. Analysing all 22 countries no correlation of high fat diet and high incidence CHD can be found.
Why We Need Cholesterol
Cholesterol is so important for us that the body produces it both in our liver and in our brain and its deficiency has a detrimental impact on every aspect of our health because it plays a critical role within our trillions of cell membranes that are continuously interacting with each other and do so thanks to cholesterol.
Cholesterol is essential for our brains. Our brains contain about 25 percent of the total cholesterol in your body. It is critical for the connections between your neurons, which means that we can think, learn and have memories.
We need cholesterol to produce steroid hormones, including your sex hormones. Vitamin D is also synthesized from a close relative of cholesterol: 7-dehydrocholesterol.
There is also strong evidence that having too little cholesterol INCREASES your risk for cancer, memory loss, Parkinson’s disease, hormonal imbalances, stroke, depression, suicide, and violent behavior.
A interesting new theory by Dr. Stephanie Seneff, of the Weston A. Price Foundation, is that cholesterol combines with sulfur to form cholesterol sulphate which helps thin your blood by serving as a reservoir for the electron donations you receive when walking barefoot on the earth. According to her theory, with this blood-thinning mechanism, cholesterol sulfate actually provides natural protection against heart disease.
The Risk Factors For Heart Disease.
Atherosclerosis is the result of a process of damage and repair mechanism that take place in your artery walls and causes blood clotting. When the wall of your arteries (endothelium) is damaged, a repair mechanism is set into motion which creates a “scab.” the endothelial wall grows over it, causing the area to become thickened. This is what is called atherosclerosis.
There’s no fat (cholesterol) clogging the artery at all. The arterial wall thickens as a result of your body’s natural repair process.
What causes damage to your arteries is sugar, especially fructose, which start the inflammation process in the arteries.
The risk factors for heart disease risk are:
– The HDL/total cholesterol ratio. This percentage should ideally be above 24 percent. Below 10 percent, it’s a significant indicator of risk for heart diseaseThe triglyceride/HDL ratio which ideally should be below 2
– The fasting blood sugar level: a level of 100-125 mg/dl indicates a nearly 300 percent increase higher risk of having coronary heart disease than for level below 79 mg/dl
– The iron level: Iron is a strong oxidative stress. Excess iron levels you can damage your blood vessels and increase your risk of heart disease. Ferritin levels should not be above 80 ng/ml. Donate your blood to lower them is the easies way to lower them.
Types Of LDL Cholesterol
The discovery of the various subtypes of cholesterol, particularly small “dense” LDL and a larger form called large “buoyant” LDL, was made already in 1950 by Dr. John Gofman, a professor of medical physics at the University of California and his discovery could have blown the lid off the cholesterol theory.
Dr. Gofman found that, in general, it was the triglyceride level (fats in the blood), not cholesterol level, that was most closely linked to heart disease and atherosclerosis.
By 1960, studies had clearly shown this to be true, yet the cholesterol proponents had the ear of the government and the media, and they were successful in drowning out their challengers.
The reason that the government and media resisted the idea that triglycerides were more important is simple: The major regulator of triglycerides in the diet is not cholesterol or fat, but sugar and refined carbohydrates. They could not admit that the high carbohydrate diet they had proposed for the past 70 years was killing people. The American Diabetes Association for decades has recommended a high carbohydrate diet for diabetics.
The discovery of Dr. Gofman also explains why diabetics, who often have normal or low levels of cholesterol, have such a high incidence of heart disease and stroke.
A diet low in saturated fats and high in sugars and refined carbohydrates lowers HDL cholesterol and raises triglycerides and VLDL cholesterol — the very pattern that is most associated with cardiovascular and stroke risk, especially among older people. BAD, VERY BAD.
Sugars and refined carbohydrates are the worst culprits of raising the deadly small dense form of LDL (particles B) while saturated fats are known to slightly increase LDL levels, BUT, saturated fats also increase the large protective LDL particles (particles A). Despite these facts having been discovered many years ago, most doctors do not require for their patients the sub-typing of cholesterol.
The small dense LDL is made of a protein called Apo B. The doctor can order a test that measures the amount of Apo B, which gives a lot more risk information than a total LDL-cholesterol test. A high-sugar, high-refined carbohydrate diet increases triglycerides and the concentration of Apo B, which is strongly associated with high cardiovascular risk.
There is a strong link between insulin resistance and atherosclerosis because high levels of insulin promotes inflammation in many tissues, including blood vessels. This inflammation is the main cause of hardening of the arteries (atherosclerosis) which is the chronic inflammation of blood vessels. The greatest increase in the incidence of diabetes is among those with insulin resistance called Type 2 diabetes.
New studies have confirmed that if you have a high-cholesterol diet but keep insulin levels very low, you do not develop significant atherosclerosis. But if you just add insulin you will develop substantial atherosclerosis.
A very concerning finding in a study about the statin drugs Lipitor, Mevacor and Pravachol, recently reported in the journal Nature Medicine, showed that statin drugs produced significant suppression of vital immune cells, the helper T-cells fundamental for protecting against cancer and fungal, bacterial and viral infections.
Chronic immune suppression in the millions of patients taking statin drugs would mean that a very high number of them would be at high risk of developing cancer, and those already having cancer would see exponential growth and spread of their cancers.
If you become immune-suppressed, you become resistant to antibiotic treatment, therefore at high risk of developing extremely hard to cure infections. And if you are a diabetic you are already immune suppressed as a result of your disease so adding statins will further lower your immunity, leading to serious damage.
Statins Linked to Breast Cancer
It has been known for some time that a high cholesterol levels reduces the risk of brest as well as most cancers. Lowering cholesterol levels increases the risk of breast cancer and of other types of cancers as well.
A large cholesterol study called the CARE study, in 1996 comparing women taking a statin drug to those not on a statin, found that 12 of 290 women in the statin group developed breast cancer compared to just 1 of 290 of the women not on the drug.
Since then, several studies have found the same link low cholesterol-higher breast cancer risk. and another study demonstrated a twofold increase in breast cancer in women taking a statin for at least 10 years.
The Women’s Health Initiative study found a 70 percent higher risk of diabetes in postmenopausal women on statin drugs. And diabetes is associated with a higher risk of breast cancer and a poorer prognosis.
Another disturbing finding is that women taking statins for 10 years or more were found to have a dramatic increase in fat weight gain, which also is strongly linked to breast cancer.
You would think that these finding would be something that all women would want and even need to know. But drug companies spend millions of dollars each year on advertising the paltry benefits reported with statin in terms of reducing heart attack, and do everything possible to bury these alarming information..
Despite all these serious concerns, the pharmaceutical companies and their friends in medical academia are now calling also for all obese people, for post-menopausal women, for people high blood pressure and for those who live sedentary lifestyles, and even for children to take the drug.
For years the recommended blood cholesterol was in the range of 200 to 210 mg/dl (milligrams/deciliter). A few years ago, the medical community in agreement with the pharmaceutical companies, pushed to lower the standard to a 150 level. That is quite understandable: statins, which are expensive and have to be taken for a lifetime, generate a huge cash flow for the pharmaceutical companies, in fact, they are now the single biggest market in the $600 billion global prescription drug business.
In order to keep their sales high, the largest statin makers, Merck, Bristol-Myers Squibb and Novartis, donate millions to major medical universities where, as a result, medical students, interns and residents in training are taught the propaganda endlessly. They also hire big- named cardiologists for “consulting work” and fund statin-related research for these universities.
So Why Take Statin Drugs.
Unoxidized cholesterol is perfectly harmless even when elevated; only elevated oxidized cholesterol causes strokes and heart attacks Studies have shown that oxidised cholesterol is correctable by dietary changes and special nutritional supplements alone, wth antioxidants and especially with flavonoids, all available through a diet high in vegetables and with supplements.
Because almost 75 percent of your cholesterol is produced by your liver, which depends on your insulin levels. If you are able to optimize your insulin levels, you will automatically optimize your cholesterol.
The primary recommendations for safely regulating your cholesterol have to do with modifying your diet and lifestyle:
I am talking of drastic lifestyle change. Adhere strictly The 19-point Guideline For General Health – there you will find everything you need to know to treat effectively and reverse your heart disease, as well as all major diseases, without dangerous prescription which will only make of you a chronic patient. (And, if you are already healthy, you will never get sick).
You Can Take Also A Little Extra Of:
– Flavonoids such as green tea extract, grape seed extract (OPCs), quercetin, luteolin and kaempferol (found in artichoke extract and ginkgo biloba), which will prevent cholesterol from oxidizing.
– A multivitamin/mineral supplement without iron in a powder form, either in a capsule or in bulk
– Magnesium ascorbate (buffered vitamin C): 1000 mg three times a day on an empty stomach between meals
– Magnesium citramate: 500 mg twice a day with meals
– L-carnitine: 500 mg twice a day on an empty stomach (improves heart strength)
– CoQ10: 150 mg a day (in olive or rice brain oil) (alternative is Idebenone, 50 mg twice a day)
– Vitamin D : 1,000 IU a day
Until next time